David A. Booth
Born (1938-08-01) 1 August 1938
England
NationalityBritish
Alma materUniversity of Oxford
Known forindividual psychology, associative learning of appetites (conditioned satiety), neuroscience of motivation, weight control, retail product choices (consumer behaviour)
Scientific career
FieldsPsychology
InstitutionsUniversity of Birmingham
Websitebirmingham.ac.uk > booth-david Archived

David Booth works full-time in research and research teaching as an honorary professor at the School of Psychology in the College of Life and Environmental Sciences of the University of Birmingham (UK). According to his Web page [1] he investigates the ways in which an individual's life works. His research and teaching centre on the processes in the mind that fit acts and reactions of human beings and animals to the passing situation.

Work

David Booth carried out work that contradicted the theory that dual centres of the hypothalamus control eating, the lateral hypothalamus for hunger [2] and the ventromedial hypothalamus for satiety [3] and began to replace it with a theory of the control of food choice and intake through learnt connections distributed around the brain.[4] With colleagues he built a simulation of the physiological and learning mechanisms influencing eating patterns in people and laboratory animals,[5] and extended it to include cultural and interpersonal influences.[6]


References

  1. ↑ Booth's personal page at the University of Birmingham ( Archived)
  2. ↑ Booth DA. Localization of the adrenergic feeding system in the rat diencephalon. Science 1967;158:515‑7. Matthews JW, Booth DA, Stolerman IP. Factors influencing feeding elicited by intracranial noradrenaline in rats. Brain Res. 1978;141:119‑28. Cp. Ungerstedt U. Acta physiol Scand 1970;80(4):35A-36A, 1971; Suppl367:97-122.
  3. ↑ Booth DA, Toates FM, Platt SV. Control system for hunger and its implications in animals and man, in D Novin, W Wyrwicka, GA Bray (eds) Hunger 1976; New York: Raven Press:127‑42. Duggan JP, Booth DA. Obesity, overeating and rapid gastric emptying in rats with ventromedial hypothalamic lesions. Science 1986;231:609‑11. Duggan JP, Booth DA. Failure to demonstrate that accelerated gastric emptying after VMH lesions is secondary to excess weight gain. Amer. J. Physiol. 1991;261:515-6.
  4. ↑ Booth DA. Vertebrate brain ribonucleic acids and memory retention. Psychol. Bull. 1967;68;149‑77. Lovett D, Goodchild P, Booth DA. Depression of intake of nutrient by association of its odor with effects of insulin. Psychon. Sci. 1968;11:27-8. Booth DA, Miller NE. Lateral hypothalamus mediated effects of a food signal on blood glucose concentration. Physiol. Behav. 1969;4:1003-9. Booth DA, Simson PC. Food preferences acquired by association with variations in amino acid nutrition. Q. J. Exp. Psychol. 1971;23:135-45. Booth DA, Lovett D, McSherry GM. Postingestive modulation of the sweetness preference gradient in the rat. J. Comp. Physiol. Psychol. 1972;78:485-512. Booth DA, Lee M, McAleavey C. Acquired sensory control of satiation in man. Br. J. Psychol. 1976;67:137-47.
  5. ↑ Toates FM, Booth DA. Control of food intake by energy supply. Nature 1974;251;710‑1. Booth DA, Mather P. Prototype model of human feeding, growth and obesity, in DA Booth (ed) Hunger models 1978;London: Academic Press 279‑322.
  6. ↑ Booth DA. A simulation model of psychobiosocial theory of human food-intake controls. Int. J. Vitamin Nutr. Res. 1988;58:55-69. Booth DA. Physiological regulation through learnt control of appetites by contingencies among signals from external and internal environments. Appetite 2008;51:433-41.
This article is issued from Wikipedia. The text is licensed under Creative Commons - Attribution - Sharealike. Additional terms may apply for the media files.